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Cellular FADD-like IL-1-converting enzyme-inhibitory protein attenuates myocardial ischemia/reperfusion injury via suppressing apoptosis and autophagy simultaneously

But the same property that makes it useful in culture is what makes any extrapolation to human physiology fraught: broad, persistent activation of a receptor that regulates growth, metabolism, and cell survival across virtually every tissue is a fundamentally different proposition from a self-limiting local pulse

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