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Another study has shown that hepatocyte-conditional NIK deficiency alleviates nonalcoholic steatohepatitis (NASH), which involves a mechanism that is independent of NF-B but involves attenuation of JAK2-STAT5 signaling axis [28]

Although the reasons for this gender-specific outcome remain unknown, our findings underscore the nuanced and complex interplay of glutathione regulation in the context of lymphomagenesis and its potential therapeutic implications

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Bioorg Med Chem 2007;15:2396402