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Collectively, FADS knockdown led to more cytotoxic and proliferative CD8 + T cells with decreased dysfunction (Fig

Expectedly, depletion of cystine or inhibition of the Xc- antiporter using synthetic small molecule inhibitors including erastin, sulfasalazine, sorafenib and even excess of glutamate triggered cystine depletion, decreased GSH level and reduced GPX4 activity, culminating in ferroptosis induction

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3.3.2 Side effects of sulfonylureas The primary adverse effect associated with sulfonylureas is hypoglycemia