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In the mitochondria, there is an increase in Cu + , which binds directly to lipid-acylated components of the TCA cycle (e.g., dihydrolipoamide S-acetyltransferase (DLAT)), leading to lipid-acylated protein aggregation and destabilization of Fe-S cluster proteins

Environmental obesogens: organotins and endocrine disruption via nuclear receptor signaling

As we age, however, we lose much of our ability to break down protein and form glutathione from individual aminos

Menkes disease is a congenital Cu deficiency disorder that leads to emphysema (Mizuno et al., 2012)

During the initial step, the catalytic site of GPx, which consists of either selenocysteine or cysteine (Fig