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Metabolic reprogramming is a defining characteristic of cancer cells, which undergo metabolic alterations to maintain tumorigenicity and survive in the presence of immune cells and chemotherapy agents 4

Thus, luteolins mechanisms of action on fibroblasts include inhibition of NF-B activation, which is crucial for inflammatory responses [244], and modulating MAPK and PI3K/Akt pathways to regulate cell adhesion and inflammation [245] and suppressing TGF-1 signaling, which is central to fibroblast-to-myofibroblast differentiation and the progression of fibrosis [19]

High-resolution mass spectrometry identified metabolites of QACR, distributed in subcutaneously transplanted tumors of nude mice post oral administration, including Deacetyl asperulosidic acid methyl ester, paeoniflorin, liquiritin, and glycyrrhizic acid

Malakar P, Singha D,Choudhury D, Shukla S

How (Rubiaceae), and Cuscuta chinensis Lam (Convolvulaceae)