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Description

In this context, glucose metabolism inhibits apoptosis by caspase inactivation through NADPH-mediated redox inactivation of cytochrome c (134)

exhibiting substantially higher stability and a deactivation constant approximately fourfold lower than that of the soluble enzyme (Jaiswal et al., 2025)

Here we provide the first holistic analysis of the current landscape of approved prodrugs using cheminformatics and data science approaches to reveal trends in prodrug development