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The overall data postulate a dual role for mutp53, in which the exogenous components and the effects of the microbiome, though gallic acid, might decide the transition from tumor-suppressive to oncogenic function, or vice versa (Kadosh et al., 2020)

For instance, research has explored its PK/PD modeling in human volunteers to establish relationships between dosage and growth hormone response [cde2]

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Regulatory T cell migration is dependent on glucokinase-mediated glycolysis