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In preclinical research settings, experimental models may examine its role in pathways associated with oxidative stress regulation, detoxification mechanisms, and cellular response systems

For example, glutamate is subsequently converted to -ketoglutarate (-KG), a key metabolite that stimulates mTORC1 via its interaction with prolyl hydroxylases, which increases the GTP loading of Rag GTPases responsible for mTORC1 activation

[DOI] [PubMed] [Google Scholar] 123.Mitchell J.P., Carmody R.J

Park HK, Kim SM, Choi CG et al (2008) Effect of trientine on manganese intoxication in a patient with acquired hepatocerebral degeneration

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