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The reaction rate is influenced by the nucleophilicity of the thiolate, resulting in the enhanced capacity of NAC for reducing disulfide bonds compared to cysteine and GSH (Noszl et al., 2000)

This programmed inflammatory cell death, orchestrated by the caspase superfamily, plays a pivotal role in pulmonary injury via canonical (e.g., caspase-1) or non-canonical pathways (Yu et al., 2021)

These findings establish innate RNA sensing as an intrinsic driver of hematopoietic stem cell aging and highlight modulation of MDA5-dependent pathways as a potential strategy to mitigate age-related hematopoietic dysfunction