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Rights and permissions About this article Cite this article Grabner, G.F., Xie, H., Schweiger, M

doi: 10.3390/microorganisms7110550 37 GottliebABDeodharAMcinnesIBBaraliakosXReichKSchreiberSet al

Anti-atherosclerotic activity Mechanism of action Atherosclerosis is driven by chronic inflammation of the arteries, particularly the aorta, where oxidized LDL (ox-LDL) and reactive oxygen species (ROS) activate and impair vascular endothelial cells

Hypoxia, activation of oncogenes, mutations in mitochondrial DNA, and loss of tumor suppressors have all been shown to lead to increased mitochondrial ROS-dependent tumorigenesis

61-63 Treatment with gamma-glutamylcysteine ethyl ester can increase intracellular reduced GSH concentrations, and has been shown to result in a dose-dependent reduction in infarct size in a canine model of occlusion-reperfusion