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To counteract this imbalance, cancer cells adapt by upregulating GSH biosynthesis, maintaining a redox equilibrium that supports survival and proliferation (Traverso et al., 2013)

Tumor necrosis factor (Tnf)-alpha inhibits insulin signaling through stimulation of the p55 tnf receptor and activation of sphingomyelinase

Once the vulnerable plaque is raptured, the thrombotic vessels, especially small diameter vessels such as coronary arteries, can be occluded

Transplantation 60 , 794798 (1995)

Endogenous Hydrogen Sulfide and ERK1/2-STAT3 Signaling Pathway May Participate in the Association between Homocysteine and Hypertension