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E.FutterM.Garcia-ArencibiaM.Green-ThompsonZ

Strategies aimed to increase cytosol GSH (e.g., NAC) may not be an optimal approach for boosting mGSH and therefore for treatment of SH or AD, as it would result in mainly increasing cytosol GSH without replenishing mGSH levels A new approach has been exploited recently in several contexts by the development of a series of cationic antioxidants targeted to mitochondria, including derivatives of the endogenous antioxidants ubiquinol (MitoQ), alpha-tocopherol (MitoVit E), and of the synthetic spin trap PBN (MitoPBN

Our data reaffirm the current push to improve poorly annotated regions of the genome, as these may be sites of variation functionally relevant to AN [11]

For instance, the QikProp module from Schrdinger software predicts key pharmacokinetic parameters such as logP, logS, Caco-2 cell permeability, serum protein binding, and human ether-a-go-go related gene (hERG)-K ion channel blocking