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Centrilobular (Figures 5459) Sometimes referred to as periacinar necrosis, it consists of irreversible cell death of centrilobular hepatocytes and is often seen after anoxia, or exposure to tannic acid, chloroform, or other hepatotoxic agents (Gopinath, Prentice, and Lewis 1987)

The new stomach pouch is so small that there is a decrease in gastric acid

This severe metabolic blockade reduces the transmitter content of individual vesicles by more than 50%, resulting in a dramatic decrease in quantal size and a rapid, dose-independent decay of the sympathomimetic response (Sulzer et al, 1995

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