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Receptor for advanced glycation end products (RAGE) is a key factor for plaque destabilization in diabetes mellitus, where its downregulation may suppress atherosclerotic plaque development, an effect mediated by NF-B inhibition (100, 101)

After tumors developed to about 400 mm 3 , we performed comparative efficacy studies by dividing mice into four groups ( n = 3 per group) to minimize weight and tumor size differences among different groups

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