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Single-cell transcriptomics and proteomics studies reveal that suppressing excessive autophagy in established obesity paradoxically improves metabolic outcomes by reducing inflammatory activation

However, inhibiting CAF autophagy effectively suppressed anti-tumor immune responses and anti-tumor efficacy in mouse models by upregulating CD274/PD-L1 levels [128, 129]

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[10] but work from our own group has shown that increased inflammatory activity is related to non-response in depressed patients treated with antidepressants [7]