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The future is promising for this therapy (Figure 6c)

The findings highlight pathways such as fibroblast-to-myofibroblast transition (FMT), redox balance regulation, lysosomal dysfunction, ETC dysfunction, autophagy, and calcium dysregulation as potential contributors to hEDS pathogenesis

Conditions such as renal impairment (creatinine clearance under 60 mL per minute or clinically significant elevated serum creatinine), anemia, diabetes mellitus, electrolyte imbalance, vitamin B deficiency, and debilitating diseases may increase the possibility of peripheral neuropathy

and certain cancers, where probiotics may help alleviate treatment-related adverse effects (3)