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Moreover, certain proteasome degradation processes are independent of lysine residues on the substrate protein (i.e., non-canonical ubiquitination) 11

moreover, it also regulates several signaling pathways, transcription factors, molecular chaperone functions, apoptosis, autophagy, and dopamine homeostasis (Skou et al., 2024)

The future of clinical research in neuroscience, therefore, lies in collecting real-world data and using them as catalysts for clinical trial design and in further development of individually-tailored treatment procedures

In AP, once mitochondrial Ca 2+ handling is overwhelmed and m collapses, mitochondrial dysfunction may secondarily promote a secretory environment favorable to premature enzyme activation through ATP depletion, defective local Ca 2+ buffering, oxidative stress, and impaired vesicular homeostasis

These interactions play pivotal roles in ovarian physiological processes and therefore warrant further comprehensive investigation