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This analysis identified that lysine 133 (K133) of IL-1 was targeted for ubiquitylation (marked by a Lys--Gly-Gly (diGly) linkage), while the adjacent serine (S134) was phosphorylated (Fig

In this section, we mainly discussed and described the negative effects of ROS during ischemiareperfusion by regulating inflammatory responses, the blood-ridge barrier, mitochondrial damage, and programmed cell death (Figure 2)