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In vivo studies in Apc min/+ mice demonstrated that deoxycholic acid (DCA) promotes the adenoma-adenocarcinoma transition and DCAinduced changes to the microbial community promoted intestinal carcinogenesis (127, 128)

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Mechanistically, baicalein demonstrably activated and cleaved caspase-12 and-3, known executioners of apoptosis, suggesting its direct engagement in the apoptotic cascade

To understand just how powerful this job is, keep in mind that the level of glutathione in cells rivals the key ingredients of life: As can be seen in Figure 1, this is the same concentration in cells as glucose, potassium, and cholesterol