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Therefore, the ultimate role of HIF-1 is the result of a complex balance between its direct cell-autonomous inhibitory effects and its indirect, microenvironment-driven pro-osteogenic effects

In parallel with tumor shrinkage, Erastin significantly upregulated the levels of 4-HNE, a lipid peroxidation marker, but not cleaved caspase-3 and Ki67, in LDHB KD tumors, reinforcing the notion that ferroptosis underpins the anti-tumor effects of SLC7A11 inhibition in LDHB KD cancer cells ( Supplementary Figure 9c, e )