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Studies have demonstrated that FSP1 can potentiate cell membrane repair and inhibit ferroptosis through the ESCRT-III-dependent membrane repair pathway, independently of CoQ10 (77)

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The results showed that the concentration of uricase reached 5 U/mL, and the cell viability decreased to only 64.1%, indicating that the H 2 O 2 generated through the degradation of UA by uricase could effectively induce cell death (Fig

This condition occurs when the tight junctions of the intestinal wall become loose, allowing toxins, bacteria, and other harmful substances to sneak through the walls and invade the bloodstream