hypothesis glutathione persister cells

hypothesis glutathione persister cells Drug-tolerant in cancer: the cutting edges and future directions Frontiers | Exogenous cystine increases

Name: hypothesis glutathione persister cells Drug-tolerant in cancer: the cutting edges and future directions Frontiers | Exogenous cystine increases
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Frontiers Exogenous cystine increases susceptibility of drug resistant Salmonella to gentamicin by promoting oxidation of glutathione metabolism and imbalance of intracellular redox levels JCI CD8+ T cells sustain antitumor response by mediating crosstalk between adenosine A2A receptor and glutathione GPX4 Persister cancer cells: Iron addiction and vulnerability to ferroptosis: Molecular Cell Mechanism of Drug Tolerant Persister Cancer Cells: The Landscape and Clinical Implication for Therapy Journal of Thoracic Oncology Vaccinia related kinase 2 inhibition elicits vulnerability of glutathione metabolism in pancreatic cancer Cell Death & Disease

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Retrieved July 18, 2009

Selective modulation of glutathione levels in human normal versus tumor cells and subsequent differential response to chemotherapy drugs

633 Chronic social defeat stress-induced depressed mice showed iron accumulation and iron homeostasis imbalance in the brain, accompanied by downregulation of GSH levels and GPX4 expression, which may be a potential mechanism of lipid peroxidation and ferroptosis in depressed mice

Molecules 26 (22), 6994 (2021)

The expression of lncRNA CRNDE negatively correlates with profibrotic genes, for example, and its overexpression has been shown to reduce cardiac fibrosis and improve contractile function in DCM mice (143)