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Mitochondrial dysfunction as a cause of axonal degeneration in multiple sclerosis patients

(2012), is characterized by iron-dependent accumulation of lipid peroxides, distinguishing it from classical cell death modalities such as apoptosis, necrosis, and autophagy, ferroptosis exhibits a stronger dependence on intracellular iron than other regulated cell death pathways

This hypothesized VEGF/VEGFR interaction may activate a complex intracellular signaling cascade, which may involve multiple proteins such as phospholipase C, protein kinase C, and the mitogen-activated protein kinases

For IBS, IBD, gastritis, or ulcers, protocols typically use oral administration at 0.5 to 1 mg daily to maximize local gastrointestinal exposure

Inoue K, Tsuda M