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These discrepancies likely reflect a non-linear relationship in which moderate mitochondrial dysfunction promotes migratory traits, whereas profound OXPHOS impairment constrains metastatic capacity in a tumor-type- and stage-dependent manner

The intrinsic factor complication adds another layer

Lack of glutathione peroxidase-1 exacerbates a beta-mediated neurotoxicity in cortical neurons

While cancer causes and treatments are very complex, it is possible that GHK-Cu, which has multiple effects on gene expression and has been shown to exhibit favorable gene effects reversing gene expression back to health in metastasis prone colon cancer gene signature and COPPD gene signature, can help slow or regress selected cancers in humans

Cohen-Kaplan, V