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In conclusion, we show that chronic in vivo GSH deficiency, despite inducing CYP2E1 and hepatic oxidative stress, triggers adaptive mechanisms that protect the liver against ethanol-induced toxicity

Increased iron load and exacerbated lipid peroxidation are key features of ferroptosis, aligning closely with the molecular biological changes observed in the brains of PD patients (de Farias et al., 2016

Given the decrease in DMV Ach neuronal activity in morphine-treated mice, we employed the chemogenetic designer receptor exclusively activated by designer drugs (DREADDS) technique to selectively activate or inhibit small intestine-projecting DMV Ach neurons

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Analogous with the majority of the previously reported oxidase-like nanozymes, pFeSAN as oxidase mimetics with oxygen as electron acceptor might generate reactive oxygen species (ROS, e.g.OH, 1 O 2 ,O 2 ) to oxidize TMB 1,2,3