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Rowson and Duma, 2013

Therefore, there is a need to develop a therapeutic strategy that can modulate complement activity after HI without completely blocking its beneficial functions in the developing brain

Postsynaptic mechanisms of excitotoxicity: involvement of postsynaptic density proteins, radicals, and oxidant molecules

Furthermore, formation of Cr-Asc (Ascorbate), Cr-GSH, and Cr-cys (cysteine) crosslinks depletes cellular antioxidants and induces oxidative stress (Yao et al., 2008)