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Therefore, reducing autoreactive Th1-type CD4 + T-cell glycolysis and/or inhibiting antioxidant activity could be potential strategies to prevent the development of T1D ( Figure 2 )

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Preliminary findings implied that the peptide may have contributed to increased body mass and a probable elevation in the overall tibial and vertebral BMC (bone mineral content) as detected by DEXA compared to the placebo group

VNN1, a potential biomarker for pancreatic cancer-associated new-onset diabetes, aggravates paraneoplastic islet dysfunction by increasing oxidative stress