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Collectively, these findings raise the possibility that the mechanism of glucocorticoids increasing serum IGF-I levels is by directly inducing post-IGF-IR signaling resistance

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Irrespective of the location and source of GrK in vivo , GrK protein could potentially be therapeutically administered in tumors to trigger sVEGFR1 release from endothelial cells to impair angiogenesis and tumor growth

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doi: 10.1128/JCM.03529-13 25 BentleyS