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venous blood vessels, and thereby does not tend to result in significant postural hypotension preferentially dilates arterioles Experimental studies indicate that its effect can be abolished by endothelial denudation, as well as pre-treatment with non-selective COX inhibitors or prostacyclin receptor antagonists, suggesting an endothelial-dependent mechanism involving COX-mediated formation of prostacyclin (Maille et al, 2016)

Osteoblast-specific knockout of the insulin-like growth factor (IGF) receptor gene reveals an essential role of IGF signaling in bone matrix mineralization

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Such cases can result from inflammation, degenerative diseases, or autoimmune diseases