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E2 interact with AF2 domain of ER and this amplifies the gene expression whereas SERM prevents this interaction and its consecutives transcriptional effects [8]

Key Features: 80120x longer half-life than native IGF-1 Binds weakly to IGFBPs remains active longer in the bloodstream Stimulates satellite cell activation key for muscle hypertrophy Promotes lean mass gain and fat loss simultaneously No direct suppression of testosterone or HPTA function Unrepeatable Insight : IGF-1 LR3 is not a growth hormone itselfbut it is the molecule through which GH exerts most of its anabolic effects

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