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In Huntingtons disease models, ALOX5 has been identified as a key factor in HTT-Q94-induced, ACSL4-independent ferroptosis, emphasizing its independent role in ferroptosis, particularly in neurodegenerative diseases (Song et al., 2023)

The 2-Cys Prxs form dimers during the reaction cycle and adopt decameric complexes, whereas the atypical 2-Cys and 1-Cys Prxs do not form dimers (as stated in previous reviews [48,49] )

Mechanistically, numerous animal experiments have revealed the protective potential of betaine, whose effects are primarily reflected in combating oxidative stress, inhibiting inflammatory responses, and regulating primary signaling pathways

10.1016/j.freeradbiomed.2023.12.031 60 JunodA

Eur Urol (2018) 74(3):37686