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"Mechanisms" people cite (and what they don't prove) You'll often see references to growth factors and vascular signaling (like pathways associated with tissue repair)

It is worth flagging early, however, that even the founding observation carried a two-sided signal: the same tripeptide that prolonged normal-cell survival also stimulated growth in neoplastic cells, a duality that anyone extrapolating to systemic use should keep in view

Known to regulate gene expression and protein synthesis, epithalon has been found to suppress both the CCl11 and HMGB1 genes, thereby increasing the lifespan of test subjects [4]

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