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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells links GSH sensing with iron metabolism: Molecular Cell Development of a mouse model

Development of a mouse model expressing a bifunctional glutathione synthesizing enzyme to study glutathione limitation in vivo Journal of Biological Chemistry glutathione and mitochondrial function SLC25A39 is necessary for mitochondrial glutathione import in mammalian cells Controlling glutathione entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease Signal Transduction and Targeted Therapy DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39 mediated mitochondrial glutathione transport ScienceDirect Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis: Molecular Cell

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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells links GSH sensing with iron metabolism: Molecular Cell Development of a mouse model

GHK-Cu is a copper peptide

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells links GSH sensing with iron metabolism: Molecular Cell Development of a mouse model

The two compounds illustrate how diverse the broader GH research peptide field is same biological axis, completely different research mechanisms

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells links GSH sensing with iron metabolism: Molecular Cell Development of a mouse model

doi: 10.1161/01.res.0000035038.41739.cb

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells links GSH sensing with iron metabolism: Molecular Cell Development of a mouse model

Carr, G

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells links GSH sensing with iron metabolism: Molecular Cell Development of a mouse model
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