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Kidney PE-ox levels are increased in experimental folic acid-AKI, where ferroptosis plays a key role, and also the urinary sediment of AKI patients, regardless of AKI cause.29 Altogether, this information suggests that ferroptosis may occur in human kidney disease, but unraveling its precise role in the course of kidney disease awaits the performance of clinical trials

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Its responsible for fast-tracking fatty acids to your mitochondria, which burn the fat into energy