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In a sciatic nerve IRI model, oral administration of lutein before ischemia and after reperfusion reduced neuropathic pain and nerve injury by decreasing oxidative stress and suppressing inflammatory mediators, such as NF- B and TNF- , while preserving endogenous antioxidant defenses [56]

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This therapy can be beneficial for patients who are unable to obtain sufficient amounts of this nutrient through dietary sources or oral supplements

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