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Although cholesterol was not directly manipulated in this study, our findings are consistent with a model in which cholesterol delivery via HDL and sequestration into lipid droplets act in parallel with triglyceride storage to suppress peroxidative stress and promote ferroptosis resistance

Views of aging and senescence frequently highlight a deterioration in mitochondrial health and function with age, and investigations of NAD+ depletion and the associated oxidative stress and damage corroborate these theories (1-2)

In still another embodiment, an ADC of the invention specifically binds more then one Eph receptor

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