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GSH acts as an electron donor to reduce lipid hydroperoxides upon GPX4 action ( S -glutathionylation should be explored in the future, since it seems to be a mechanism of redox switch in ECs accounting for vascular homeostasis ( PTGS2 , GPX4 and GSS was also assessed ( Figure 1E ) and promotes the increase of intracellular ROS ( Figure 1A ) and lipid peroxide levels ( Figure 1C ), without affecting the mitochondrial ROS content ( Figure 1B )

Clinical features of Parkinson disease when onset of diabetes came first: a case-control study

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Neuronal Loss of the Glutamate Transporter GLT1 Promotes Excitotoxic Injury in the Hippocampus. Frontiers in Cellular Neuroscience 15: 788262