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Maintenance of Fe-S clusters is antagonistic to survival under cystine starvation To determine if the GSH-dependent maintenance of Fe-S protein function plays a contributing role to ferroptosis in NSCLC, we evaluated cell viability under cystine starvation in response to the disruption of Fe-S cluster homeostasis

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According to Sev transcriptional sequencing, the expression of ferroptosis- and mitophagy-related gene-activating transcription factor 4 (ATF4) was enhanced, and Sev induced ferroptosis in glioma cells by activating the ATF4-CHAC1 pathway (Xu et al., 2022)