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About this article Cite this article Tollenaere, J., Wauters, T

The nephrotoxic mechanisms of IS have been characterized primarily through in vitro and animal models, with limited validation in human DKD

The loss of topoisomerase II or its inhibition by fluoroquinolones results in accumulation of positively supercoiled mtDNA, followed by cessation of mitochondrial transcription and replication initiation, causing depletion of mtDNA copy number