slc25a39 is necessary for mitochondrial glutathione import in mammalian cells

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Controlling glutathione entry into mitochondria:

Name: slc25a39 is necessary for mitochondrial glutathione import in mammalian cells Controlling glutathione entry into mitochondria:
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Controlling glutathione entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease Signal Transduction and Targeted Therapy Slc25a39 and Slc25a40 Expression in Mice with Bile Duct Ligation or Lipopolysaccharide Treatment Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis: Molecular Cell Up regulated SLC25A39 promotes cell growth and metastasis via regulating ROS production in colorectal cancer Mitochondrial Glutathione in Cellular Redox Homeostasis and Disease Manifestation

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It helps to neutralize free radicals, which can lead to neurodegeneration and neurological disorders such as Alzheimer disease, Parkinson disease, and Huntington disease

No features of massive fibrosis, persistent metanephric duct, cartilage/osseous metaplasia, or other structures typical for renal dysplasia were present in the kidney