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Also, ferroptosis contributes to the accentuated myocardial remodeling and more severe dysfunction after MI by triggering inflammatory responses (Huang and Frangogiannis 2018)

The oxidation of fatty acids is affected as a result of the metabolism of fructose by fructokinase C, thus the result of the consumption of adenosin triphosphate (ATP), the production of uric acid and the nucleotide exchange, and finally, concomitant mitochondrial dysfunction [55]

Ferroptosis as a novel therapeutic target for Friedreichs ataxia

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Owing to its high molecular weight (~ 5700 kDa) and hydrophilic properties, insulin has a slow diffusion rate across the epithelial layer [8]