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igf-1 lr3 2017

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Description

The phenotypic hallmark of the SOD1-dependent ALS is the presence of amyloid aggregates of SOD1 in affected tissues, which are the ultimate outcome of SOD1 misfolding the product of a gradual destabilization of the SOD1 structure due to the loss of metal ions and the reduction of the stabilizing intrasubunit disulfide bond 10,11,12

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Erenberk U, Dundaroz R, Gok O et al (2014) Melatonin attenuates phenytoin sodium-induced DNA damage

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This will make GPX4 enzyme hungry and make it unable to neutralize lipid peroxidation caused by iron overload

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