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Mechanistically, a key way MOTS-c influences fat metabolism is through its upstream activation of AMPK and downstream effects on gene expression

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As well, the use of dopamine antagonists, which not only lowers the prolactin levels but may have independent metabolic properties of their own, lead to divergent results

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In addition, we have shown that BL toxicity strongly inhibits ovarian development and exerts persistent suppressive effects even in the absence of lethality (Supplementary Fig

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Let's be perfectly clear: getting this wrong can compromise an entire line of study

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