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The mechanism of Met's anti-tumor effect may be that it directly reduces the production of ATP due to its function as an inhibitor of mitochondrial electron transfer chain complex I and an activator of AMPK (132)

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Though it is difficult to ascertain the exact figure of ineligible patients due to the complex criteria of assessing patient eligibility [124], creating less toxic conditioning regimens using immunotherapy could broaden patients eligibility for HCT, therefore increasing probabilities of survival