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Astrocyte Kir4.1 ion channel deficits contribute to neuronal dysfunction in Huntingtons disease model mice

10.1002/hep.23660 Hepatology 59 HuangY.DuJ.MiY.LiT.GongY.OuyangH.et al (2018)

10.3748/wjg.v23.i33.6164 62

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Uncoupled endothelial NOS, nicotinamide adenine dinucleotide phosphate hydrogen (NADPH) oxidase, xanthine oxidase, and mitochondrial electron transport are the main sources of ROS