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Although it is clear that acyl-ghrelin activates GH secretion when injected into mice and men, the specific contribution of acyl-ghrelin to physiological pulsatile GH release is less clear

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Karin, O., Agrawal, A., Porat, Z., Krizhanovsky, V

It may indicate the increased SELENOP transport across the neural barriers, due to high expression of LRP8 at the BBB, which keeps Se levels relatively stable, even during deficiency periods making an adequate Se pool available in the CNS (Zachara et al., 2001)

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