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This arrangement places GHK-Cus copper-binding strength in the same general range as the labile copper pool carried by albumin in plasma which is precisely why the peptide can plausibly participate in physiological copper exchange rather than either locking copper away permanently or dumping it uncontrollably

Studies in models of spinal cord injury, traumatic brain injury, and peripheral nerve damage have shown protective effects

The WB results showed that AMSCs treatment exhibited a restoration in liver OXPHOS subunits expression that was impaired by overdosed APAP (Fig