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doi: 10.2147/OTT.S50208

GPX4 must use GSH as a substrate to eliminate intracellular lipid ROS and maintain redox equilibrium

In this model, PINK1 or Parkin deficiency disrupts mitophagy, resulting in excessive mROS production, mitochondrial damage, increased TGF-1 expression and Drp1 recruitment in RTECs, all of which promote renal fibrosis