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Ser/gly pathway metabolites are required for RT recovery in NSCLC To validate the observations in the plasma of patients with NSCLC and confirm our hypothesis of augmented ser/gly pathway dependency, we performed 13 C 6 -glucose tracing and metabolomics analysis in a panel of 5 NSCLC cell lines, i.e., NCI-H1299, NCI-H125, NCI-H1975, Calu-6 and NCI-H520 to identify the RT related metabolic changes

Article ID 794643, 10 pages

Switzerland: Springer (2022)

Meanwhile, PCEF@Fe employs a cooperative strategy: it chelates copper to weaken cellular defenses while simultaneously releasing iron to initiate ferroptosis, illustrating a novel approach for the coordinated targeting of multiple cell death pathways (Hua et al., 2025)

These findings support the hypothesis that pyroptosis may be involved in melanocyte death associated with ROS in vitiligo [12, 60]