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Justin Conner in studying the effects of mutations to Peroxiredoxin-6 (PRDX6) on oxidative stress in vitro and in vivo models

Administration of BmK IT2 markedly prolonged the latency to SE onset, attenuated SE severity, and suppressed hippocampal c-Fos expression during epileptogenesis, suggesting its role in modulating neuronal hyperexcitability (Zhao et al., 2008)